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This historic book may have numerous typos and missing text. Purchasers can download a free scanned copy of the original book (without typos) from the publisher. Not indexed. Not illustrated. 1921 Excerpt: ...now in the manufacture of anilin dyes is caused by the accidental evolution of hydrogen arsenide (AsH3) which may occur in connection with certain processes, especially acid reduction, and alkaline reduction when followed by acidification. These processes are described fully in a later section (see p. 53). It is a subtle and powerful poison, causing extensive destruction of the elements of the blood and, as a consequence of this, an acute degeneration of liver and kidneys. When the exposure to the fumes is great enough to set up acute poisoning, the man is seized with symptoms which may suggest, and, in fact, probably often do suggest, acute anilin poisoning. He is nauseated, complains of cramps in the stomach, vomits, complains of headache, dizziness, and faintness. The urine is dark, another resemblance to anilin poisoning. But the condition lasts longer than that caused by anilin, or even by dinitrobenzene. The case does not clear up, but grows worse, the urine becomes bloody, anemia is profound, jaundice appears, and signs of inflammation of the liver. The man may pass into a typhoid condition lasting for days, or he may die within a few days, deeply jaundiced, with constant vomiting of greenish fluid, with strangury and bloody urine. Autopsy then shows hemorrhagic inflammation of liver and kidneys. This form of poisoning has been recognized in Germany for a good many years and carefully guarded against. It has also been studied recently by physicians of the British Factory Inspection Department and by physicians attached to the British works. According to these authorities, it is only prudent to take into consideration the possibility of arsenical poisoning in any case of supposed poisoning by a nitro or amido compound when the symptoms do not clear up...

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